G-Protein Coupled Receptor 98 (GPR98) Antibody
351€ (100 µg)
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935106861
info@markelab.com
name
G-Protein Coupled Receptor 98 (GPR98) Antibody
category
Primary Antibodies
provider
Abbexa
reference
abx376138
tested applications
IHC
Description
G-Protein Coupled Receptor 98 (GPR98) Antibody is a Rabbit Polyclonal antibody against G-Protein Coupled Receptor 98 (GPR98).
Documents del producto
Instrucciones
Data sheet
Product specifications
| Category | Primary Antibodies |
| Immunogen Target | G-Protein Coupled Receptor 98 (GPR98) |
| Host | Rabbit |
| Reactivity | Human, Mouse |
| Recommended Dilution | Optimal dilutions/concentrations should be determined by the end user. |
| Clonality | Polyclonal |
| Conjugation | Unconjugated |
| Purification | Purified by affinity chromatography using epitope-specific immunogen. |
| Size 1 | 100 µg |
| Form | Liquid |
| Tested Applications | IHC |
| Buffer | PBS, pH 7.4, containing 0.02% sodium azide and 50% glycerol. |
| Availability | Shipped within 5-12 working days. |
| Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| UniProt ID | Q8WXG9 |
| Gene ID | 84059 |
| Alias | ADGRV1,FEB4 GPR98,MASS1,USH2B,USH2C,VLGR1,VLGR1b |
| Background | Antibody anti-ADGRV1 |
| Status | RUO |
| Note | Concentration: 1 mg/ml - |
Descripción
Adhesion G-protein coupled receptor V1 (ADGRV1), also known as GPR98, is a member of the adhesion GPCR family. ADGRV1 is a large transmembrane protein with an extracellular domain containing multiple adhesion motifs, a seven-transmembrane domain characteristic of G protein-coupled receptors (GPCRs), and an intracellular domain involved in signaling. ADGRV1 is primarily expressed in the inner ear and is essential for auditory function. It plays a crucial role in the development and maintenance of stereocilia, the specialized hair-like structures in the inner ear that detect sound vibrations and convert them into electrical signals. Mutations in the ADGRV1 gene are associated with various forms of hereditary hearing loss, including Usher syndrome type IIC and nonsyndromic deafness. These mutations disrupt the normal function of ADGRV1, leading to structural abnormalities in the stereocilia or impairment of signal transduction in the inner ear.
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