Cell Death-Inducing DFFA-Like Effector B (CIDEB) Antibody

Este producto es parte de CIDEB - Cell death-inducing DFFA-like effector b
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52€ (10 µg)

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935106861
info@markelab.com
name
Cell Death-Inducing DFFA-Like Effector B (CIDEB) Antibody
category
Primary Antibodies
provider
Abbexa
reference
abx014016
tested applications
ELISA, WB, IHC, IF/ICC

Description

Rabbit polyclonal antibody against CIDEB protein. Immunogen region is Internal.

Documents del producto

Instrucciones
Data sheet
Descargar

Product specifications

Category
Primary Antibodies
Immunogen Target
Cell Death-Inducing DFFA-Like Effector B (CIDEB)
Host
Rabbit
Reactivity
Human, Mouse
Recommended Dilution
WB: 1/500 - 1/3000, IHC: 1/50 - 1/100, IF/ICC: 1/100 - 1/500, ELISA: 1/40000. Optimal dilutions/concentrations should be determined by the end user.
Clonality
Polyclonal
Conjugation
Unconjugated
Isotype
IgG
Purification
Purified from rabbit antiserum by affinity chromatography using epitope-specific immunogen.
Size 1
10 µg
Size 2
100 µg
Size 3
200 µg
Size 4
300 µg
Size 5
1 mg
Form
Liquid
Tested Applications
ELISA, WB, IHC, IF/ICC
Buffer
PBS (without Mg<sup>2+</sup> and Ca<sup>2+</sup>), pH 7.4, 150 mM NaCl, 0.02% sodium azide, 50% glycerol.
Availability
Shipped within 5-10 working days.
Storage
Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles.
Dry Ice
No
UniProt ID
Q9UHD4
Alias
CIDEB, cell death-inducing DFFA-like effector b, cell death inducing DFFA like effector b,CIDE-B
Background
Antibody anti-CIDEB
Status
RUO
Note
Concentration: 1 mg/ml -

Descripción

CIDEB is a protein critical for lipid metabolism and apoptosis, primarily expressed in the liver and associated with lipid droplet formation and triglyceride metabolism. It regulates lipid droplet growth and lipoprotein secretion, influencing hepatic lipid storage and export. CIDEB also participates in apoptosis by promoting the activation of caspase-dependent pathways, contributing to programmed cell death under stress conditions. Dysregulation of CIDEB has been implicated in liver diseases such as steatosis, non-alcoholic fatty liver disease (NAFLD), and liver fibrosis. Targeting CIDEB-mediated pathways is being explored for the treatment of metabolic liver diseases and conditions associated with altered lipid metabolism.
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