RAB32, Member RAS Oncogene Family (RAB32) Antibody

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Description
RAB32, Member RAS Oncogene Family (RAB32) Antibody is a Rabbit Polyclonal antibody against RAB32, Member RAS Oncogene Family (RAB32).
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Product specifications
Category | Primary Antibodies |
Immunogen Target | RAB32, Member RAS Oncogene Family (RAB32) |
Host | Rabbit |
Reactivity | Human, Mouse |
Recommended Dilution | Optimal dilutions/concentrations should be determined by the end user. |
Clonality | Polyclonal |
Conjugation | Unconjugated |
Purification | Purified by affinity chromatography using epitope-specific immunogen. |
Size 1 | 100 µg |
Form | Liquid |
Tested Applications | ELISA, WB |
Buffer | PBS, pH 7.4, containing 0.02% sodium azide and 50% glycerol. |
Availability | Shipped within 5-12 working days. |
Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
Dry Ice | No |
UniProt ID | Q13637 |
Gene ID | 10981 |
Alias | Ras-related protein Rab-32 |
Background | Antibody anti-RAB32 |
Status | RUO |
Note | Concentration: 1 mg/ml - |
Descripción
RAB32, member RAS oncogene family (RAB32) is a GTPase involved in the regulation of mitochondrial function and intracellular vesicle trafficking RAB32 has been shown to play a role in mitochondrial dynamics, particularly in the sorting and distribution of proteins to mitochondria, which are crucial for cellular energy production and metabolism In addition to its role in mitochondrial maintenance, RAB32 also participates in the regulation of vesicular trafficking within the cell, ensuring proper sorting of proteins and lipids to various cellular compartments RAB32’s function is essential for maintaining cellular homeostasis by coordinating processes such as autophagy, vesicle recycling, and intracellular protein degradation Furthermore, RAB32 has been implicated in the regulation of cell signaling, especially in the context of cell survival and response to stress RAB32’s dysfunction can contribute to diseases such as neurodegenerative disorders and cancer, where impaired cellular trafficking and mitochondrial dysfunction lead to cellular stress and uncontrolled growth
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