Non-Homologous End-Joining Factor 1 (NHEJ1) Antibody

Product Graph
312€ (60 µl)

Por favor contáctenos para obtener información detallada sobre el precio y disponibilidad.

935106861
info@markelab.com
name
Non-Homologous End-Joining Factor 1 (NHEJ1) Antibody
category
Primary Antibodies
provider
Abbexa
reference
abx003801
tested applications
WB

Description

NHEJ1 Antibody is a Rabbit Polyclonal antibody against NHEJ1. Double-strand breaks in DNA result from genotoxic stresses and are among the most damaging of DNA lesions. This gene encodes a DNA repair factor essential for the nonhomologous end-joining pathway, which preferentially mediates repair of double-stranded breaks. Mutations in this gene cause different kinds of severe combined immunodeficiency disorders.

Documents del producto

Instrucciones
Data sheet
Descargar

Product specifications

Category
Primary Antibodies
Immunogen Target
Non-Homologous End-Joining Factor 1 (NHEJ1)
Host
Rabbit
Reactivity
Human, Mouse
Recommended Dilution
WB: 1/500 - 1/2000. Optimal dilutions/concentrations should be determined by the end user.
Clonality
Polyclonal
Conjugation
Unconjugated
Isotype
IgG
Purification
Purified by affinity chromatography.
Size 1
60 µl
Size 2
120 µl
Size 3
200 µl
Form
Liquid
Tested Applications
WB
Buffer
PBS, pH 7.3, containing 0.02% sodium azide, 50% glycerol.
Availability
Shipped within 5-10 working days.
Storage
Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles.
Dry Ice
No
UniProt ID
Q9H9Q4
Gene ID
79840
NCBI Accession
NP_079058.1
Background
Antibody anti-NHEJ1
Status
RUO
Note
Concentration: 1 mg/ml -

Descripción

Related Products

FNab09545

NHEJ1 antibody

Ver Producto
P9338

Recombinant Human NHEJ1

Ver Producto
abx003801

Non-Homologous End-Joining Factor 1 (NHEJ1) Antibody

NHEJ1 Antibody is a Rabbit Polyclonal antibody against NHEJ1. Double-strand breaks in DNA result from genotoxic stresses and are among the most damaging of DNA lesions. This gene encodes a DNA repair factor essential for the nonhomologous end-joining pathway, which preferentially mediates repair of double-stranded breaks. Mutations in this gene cause different kinds of severe combined immunodeficiency disorders.

Ver Producto