Histone Acetyltransferase KAT2A (KAT2A) Antibody

312€ (60 µl)
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935106861
info@markelab.com
name
Histone Acetyltransferase KAT2A (KAT2A) Antibody
category
Primary Antibodies
provider
Abbexa
reference
abx001825
tested applications
IHC, IF/ICC
Description
KAT2A Antibody is a Rabbit Polyclonal antibody against KAT2A. KAT2A, or KAT2A, is a histone acetyltransferase (HAT) that functions primarily as a transcriptional activator. It also functions as a repressor of NF-kappa-B (see MIM 164011) by promoting ubiquitination of the NF-kappa-B subunit RELA (MIM 164014) in a HAT-independent manner (Mao et al., 2009.
Documents del producto
Instrucciones
Data sheet
Product specifications
| Category | Primary Antibodies |
| Immunogen Target | Histone Acetyltransferase KAT2A (KAT2A) |
| Host | Rabbit |
| Reactivity | Human, Mouse, Rat |
| Recommended Dilution | IHC-P: 1/50 - 1/200, IF/ICC: 1/50 - 1/200. Not tested in IHC-F. Optimal dilutions/concentrations should be determined by the end user. |
| Clonality | Polyclonal |
| Conjugation | Unconjugated |
| Isotype | IgG |
| Purification | Purified by affinity chromatography. |
| Size 1 | 60 µl |
| Size 2 | 120 µl |
| Size 3 | 200 µl |
| Form | Liquid |
| Tested Applications | IHC, IF/ICC |
| Buffer | PBS, pH 7.3, containing 0.02% sodium azide, 50% glycerol. |
| Availability | Shipped within 5-10 working days. |
| Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| UniProt ID | Q92830 |
| Gene ID | 2648 |
| NCBI Accession | NP_066564.2 |
| OMIM | 602301 |
| Background | Antibody anti-KAT2A |
| Status | RUO |
| Note | Concentration: 1 mg/ml - |
Descripción
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Histone Acetyltransferase KAT2A (KAT2A) Antibody
KAT2A Antibody is a Rabbit Polyclonal antibody against KAT2A. KAT2A, or KAT2A, is a histone acetyltransferase (HAT) that functions primarily as a transcriptional activator. It also functions as a repressor of NF-kappa-B (see MIM 164011) by promoting ubiquitination of the NF-kappa-B subunit RELA (MIM 164014) in a HAT-independent manner (Mao et al., 2009.
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