Glucagon-Like Peptide 2 Receptor (GLP2R) Antibody
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Description
GLP2R Antibody is a Rabbit Polyclonal against GLP2R.
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Product specifications
| Category | Primary Antibodies |
| Immunogen Target | Glucagon-Like Peptide 2 Receptor (GLP2R) |
| Host | Rabbit |
| Reactivity | Human, Mouse |
| Recommended Dilution | ELISA: 1/2000 - 1/5000, WB: 1/500 - 1/2000, IHC: 1/25 - 1/100. Optimal dilutions/concentrations should be determined by the end user. |
| Clonality | Polyclonal |
| Conjugation | Unconjugated |
| Isotype | IgG |
| Purification | Antigen Affinity Chromatography. |
| Size 1 | 50 µl |
| Size 2 | 100 µl |
| Form | Liquid |
| Tested Applications | ELISA, WB, IHC |
| Buffer | PBS, pH 7.4, containing 0.05% NaN3 and 40% Glycerol. |
| Availability | Shipped within 5-10 working days. |
| Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| UniProt ID | O95838 |
| Gene ID | 9340 |
| Alias | GLP2R,GLP-2R |
| Background | Antibody anti-GLP2R |
| Status | RUO |
Descripción
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Glucagon-Like Peptide 2 Receptor (GLP2R) Antibody
GLP2R Antibody is a Rabbit Polyclonal antibody against GLP2R. The GLP2 receptor (GLP2R) is a G protein-coupled receptor superfamily member closely related to the glucagon receptor ans GLP1 receptor. Glucagon-like peptide-2 (GLP2) is a 33-amino acid proglucagon-derived peptide produced by intestinal enteroendocrine cells. Like glucagon-like peptide-1 (GLP1) and glucagon itself, it is derived from the proglucagon peptide encoded by the GCG gene. GLP2 stimulates intestinal growth and upregulates villus height in the small intestine, concomitant with increased crypt cell proliferation and decreased enterocyte apoptosis. Moreover, GLP2 prevents intestinal hypoplasia resulting from total parenteral nutrition. GLP2R, a G protein-coupled receptor superfamily member is expressed in the gut and closely related to the glucagon receptor (GCGR) and the receptor for GLP1 (GLP1R).
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Glucagon-Like Peptide 2 Receptor (GLP2R) Antibody
GLP2R Antibody is a Rabbit Polyclonal against GLP2R.
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