Acyl-CoA Dehydrogenase Family Member 9 (ACAD9) Antibody (HRP)
169€ (20 µg)
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Name
Acyl-CoA Dehydrogenase Family Member 9 (ACAD9) Antibody (HRP)
Category
Primary Antibodies
Provider
Abbexa
Reference
abx317722
Tested Applications
ELISA
Description
ACAD9 Antibody (HRP) is a Rabbit Polyclonal against ACAD9 conjugated to HRP.
Documentos del producto
Instrucciones
Data sheet
Especificaciones del producto
| Category | Primary Antibodies |
| Immunogen Target | Target: Acyl-CoA Dehydrogenase Family Member 9 (ACAD9) Immunogen: Recombinant Human Acyl-CoA dehydrogenase family member 9, mitochondrial protein (1-270A). |
| Host | Rabbit |
| Reactivity | Human |
| Recommended Dilution | Optimal dilutions/concentrations should be determined by the end user. |
| Clonality | Polyclonal |
| Conjugation | HRP |
| Isotype | IgG |
| Purity | > 95% |
| Purification | Purified by Protein G. |
| Size 1 | 20 µg |
| Size 2 | 50 µg |
| Size 3 | 100 µg |
| Size 4 | 200 µg |
| Size 5 | 1 mg |
| Form | Liquid |
| Tested Applications | ELISA |
| Buffer | 0.01 M PBS, pH 7.4, 0.03% Proclin-300 and 50% Glycerol. |
| Availability | Shipped within 5-10 working days. |
| Storage | Aliquot and store at -20°C. Avoid exposure to light. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| UniProt ID | Q9H845 |
| Gene ID | 28976 |
| OMIM | 611103 |
| Alias | MC1DN20,NPD002 |
| Background | Antibody anti-ACAD9 |
| Status | RUO |
| Note | THIS PRODUCT IS FOR RESEARCH USE ONLY. NOT FOR USE IN DIAGNOSTIC, THERAPEUTIC OR COSMETIC PROCEDURES. NOT FOR HUMAN OR ANIMAL CONSUMPTION. |
Background
ACAD9 is a mitochondrial enzyme essential for the β-oxidation of long-chain fatty acids and the assembly of mitochondrial respiratory chain complex I. It functions as a dehydrogenase, catalyzing the first step in the oxidation of long-chain acyl-CoA substrates, generating energy and maintaining lipid homeostasis. ACAD9 is also a critical assembly factor for complex I, facilitating the proper formation and function of this key component of the electron transport chain. Its dual role in fatty acid oxidation and complex I assembly makes ACAD9 indispensable for mitochondrial energy metabolism. Mutations in the ACAD9 gene are associated with complex I deficiency, a severe mitochondrial disorder that can lead to cardiomyopathy, encephalopathy, and metabolic crises. The high expression of ACAD9 in energy-demanding tissues such as the heart, liver, and brain reflects its importance in maintaining mitochondrial function and energy production, positioning it as a vital enzyme in cellular metabolism.
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