Acyl-CoA Dehydrogenase Family Member 11 (ACAD11) Antibody

260€ (50 µl)
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935106861
info@markelab.com
name
Acyl-CoA Dehydrogenase Family Member 11 (ACAD11) Antibody
category
Primary Antibodies
provider
Abbexa
reference
abx242493
tested applications
ELISA, WB
Description
ACAD11 Antibody is a Rabbit Polyclonal against ACAD11.
Documents del producto
Instrucciones
Data sheet
Product specifications
Category | Primary Antibodies |
Immunogen Target | Acyl-CoA Dehydrogenase Family Member 11 (ACAD11) |
Host | Rabbit |
Reactivity | Human, Rat |
Recommended Dilution | ELISA: 1/1000 - 1/2000, WB: 1/200 - 1/1000. Optimal dilutions/concentrations should be determined by the end user. |
Clonality | Polyclonal |
Conjugation | Unconjugated |
Isotype | IgG |
Purification | Antigen Affinity Chromatography. |
Size 1 | 50 µl |
Size 2 | 100 µl |
Form | Liquid |
Tested Applications | ELISA, WB |
Buffer | PBS, pH 7.4, containing 0.05% NaN3 and 40% Glycerol. |
Availability | Shipped within 5-10 working days. |
Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
Dry Ice | No |
UniProt ID | Q709F0 |
Gene ID | 84129 |
Alias | ACAD-11 |
Background | Antibody anti-ACAD11 |
Status | RUO |
Descripción
ACAD11 is a mitochondrial enzyme that plays a critical role in the β-oxidation of long-chain fatty acids. It catalyzes the first step in the breakdown of these fatty acids, converting them into acyl-CoA intermediates that are further processed to generate energy. ACAD11 is expressed in metabolically active tissues such as the liver, heart, and skeletal muscle, which rely heavily on fatty acid oxidation for energy production. Beyond its role in energy metabolism, ACAD11 may also contribute to lipid signaling pathways, influencing cellular stress responses and metabolic adaptations. Dysregulation of ACAD11 has been associated with lipid accumulation, mitochondrial dysfunction, and metabolic disorders such as fatty liver disease. Its enzymatic specificity for long-chain substrates highlights its importance in maintaining lipid and energy homeostasis, making it a potential target for metabolic disease research.
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