Zinc Phosphodiesterase ELAC Protein 2 (ELAC2) Antibody
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Description
Zinc Phosphodiesterase ELAC Protein 2 (ELAC2) Antibody is a Rabbit polyclonal antibody for the detection of Human Zinc Phosphodiesterase ELAC Protein 2 (ELAC2).
Documents del producto
Product specifications
| Category | Primary Antibodies |
| Immunogen Target | Zinc Phosphodiesterase ELAC Protein 2 (ELAC2) |
| Host | Rabbit |
| Reactivity | Human |
| Recommended Dilution | Optimal dilutions/concentrations should be determined by the end user. |
| Clonality | Polyclonal |
| Conjugation | Unconjugated |
| Isotype | IgG |
| Size 1 | 50 µg |
| Size 2 | 100 µg |
| Form | Liquid |
| Tested Applications | ELISA, WB, IHC |
| Buffer | 0.01 M PBS, pH 7.4, 50% glycerol, 0.05% Proclin-300. |
| Availability | Shipped within 5-12 working days. |
| Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| UniProt ID | Q9BQ52 |
| Gene ID | 60528 |
| NCBI Accession | NP_001159434.1 |
| Background | Antibody anti-ELAC2 |
| Status | RUO |
Descripción
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The protein encoded by this gene has a C-terminal domain with tRNA 3′ processing endoribonuclease activity, which catalyzes the removal of the 3' trailer from precursor tRNAs. The protein also interacts with activated Smad family member 2 (Smad2) and its nuclear partner forkhead box H1 (also known as FAST-1), and reduced expression can suppress transforming growth factor-beta induced growth arrest. Mutations in this gene result in an increased risk of prostate cancer. Multiple transcript variants encoding different isoforms have been found for this gene.
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Zinc Phosphodiesterase ELAC Protein 2 (ELAC2) Antibody
ELAC2 Antibody is a Rabbit Polyclonal antibody against ELAC2. The protein encoded by this gene has a C-terminal domain with tRNA 3′ processing endoribonuclease activity, which catalyzes the removal of the 3' trailer from precursor tRNAs. The protein also interacts with activated Smad family member 2 (Smad2) and its nuclear partner forkhead box H1 (also known as FAST-1), and reduced expression can suppress transforming growth factor-beta induced growth arrest. Mutations in this gene result in an increased risk of prostate cancer. Multiple transcript variants encoding different isoforms have been found for this gene.
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