Tumor Necrosis Factor Receptor Superfamily Member 25 (TNFRSF25) Antibody
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Description
Tumor Necrosis Factor Receptor Superfamily Member 25 (TNFRSF25) Antibody is a Mouse Monoclonal antibody for the detection of Tumor Necrosis Factor Receptor Superfamily Member 25 (TNFRSF25)
Documents del producto
Product specifications
| Category | Primary Antibodies |
| Immunogen Target | Tumor Necrosis Factor Receptor Superfamily Member 25 (TNFRSF25) |
| Host | Mouse |
| Clonality | Monoclonal |
| Conjugation | Unconjugated |
| Isotype | IgG2a Lambda |
| Clone ID | O404 |
| Purity | > 95% |
| Purification | Purified by Protein A/G affinity chromatography. |
| Size 1 | 50 µg |
| Size 2 | 100 µg |
| Form | Liquid |
| Tested Applications | FCM |
| Buffer | 0.01 M PBS, pH 7.4. |
| Availability | Shipped within 5-12 working days. |
| Storage | Store at 4 °C if the entire vial will be used within 1 week. Aliquot and store at -20 °C or -80 °C for long-term storage. Avoid repeated freeze-thaw cycles. |
| Dry Ice | No |
| UniProt ID | Q93038 |
| Gene ID | 8718 |
| NCBI Accession | NP_001034753.1 |
| Background | Antibody anti-TNFRSF25 |
| Status | RUO |
| Note | Concentration: 1.4 mg/ml - |
Descripción
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Tumor Necrosis Factor Receptor Superfamily, Member 25 (TNFRSF25) Antibody
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor is expressed preferentially in the tissues enriched in lymphocytes, and it may play a role in regulating lymphocyte homeostasis. This receptor has been shown to stimulate NF-kappa B activity and regulate cell apoptosis. The signal transduction of this receptor is mediated by various death domain containing adaptor proteins. Knockout studies in mice suggested the role of this gene in the removal of self-reactive T cells in the thymus. Multiple alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported, most of which are potentially secreted molecules. The alternative splicing of this gene in B and T cells encounters a programmed change upon T-cell activation, which predominantly produces full-length, membrane bound isoforms, and is thought to be involved in controlling lymphocyte proliferation induced by T-cell activation.
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