Serum/Glucocorticoid Regulated Kinase 2 (SGK2) Antibody

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Description
Serum/Glucocorticoid Regulated Kinase 2 (SGK2) Antibody is a Rabbit Polyclonal antibody against Serum/Glucocorticoid Regulated Kinase 2 (SGK2).
Documents del producto
Product specifications
Category | Primary Antibodies |
Immunogen Target | Serum/Glucocorticoid Regulated Kinase 2 (SGK2) |
Host | Rabbit |
Reactivity | Human, Mouse, Rat |
Recommended Dilution | Optimal dilutions/concentrations should be determined by the end user. |
Clonality | Polyclonal |
Conjugation | Unconjugated |
Purification | Purified by affinity chromatography using epitope-specific immunogen. |
Size 1 | 100 µg |
Form | Liquid |
Tested Applications | ELISA, WB |
Buffer | PBS, pH 7.4, containing 0.02% sodium azide and 50% glycerol. |
Availability | Shipped within 5-12 working days. |
Storage | Aliquot and store at -20°C. Avoid repeated freeze/thaw cycles. |
Dry Ice | No |
UniProt ID | Q9HBY8 |
Gene ID | 10110 |
Background | Antibody anti-SGK2 |
Status | RUO |
Note | Concentration: 1 mg/ml - |
Descripción
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SGK2 antibody
Serine/threonine-protein kinase which is involved in the regulation of a wide variety of ion channels, membrane transporters, cell growth, survival and proliferation. Up-regulates Na(+) channels: SCNN1A/ENAC, K(+) channels: KCNA3/Kv1.3, KCNE1 and KCNQ1, amino acid transporter: SLC6A19, glutamate transporter: SLC1A6/EAAT4, glutamate receptors: GRIA1/GLUR1 and GRIK2/GLUR6, Na(+)/H(+) exchanger: SLC9A3/NHE3, and the Na(+)/K(+) ATPase.
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Serum/Glucocorticoid Regulated Kinase 2 (SGK2) Antibody
SGK2, a Ser/Thr protein kinase, is similar to serum and glucocorticoid-induced protein kinase (SGK), but this gene product is not induced by serum or glucocorticoids. Expression is induced in response to signals that activate phosphatidylinositol 3-kinase, which is also true for SGK.
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Serum/Glucocorticoid Regulated Kinase 2 (SGK2) Antibody
SGK2, a Ser/Thr protein kinase, is similar to serum and glucocorticoid-induced protein kinase (SGK), but this gene product is not induced by serum or glucocorticoids. Expression is induced in response to signals that activate phosphatidylinositol 3-kinase, which is also true for SGK.
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