NADH:Ubiquinone Oxidoreductase Core Subunit S7 (NDUFS7) Peptide
195€ (100 µg)
Por favor contáctenos para obtener información detallada sobre el precio y disponibilidad.
935106861
info@markelab.com
name
NADH:Ubiquinone Oxidoreductase Core Subunit S7 (NDUFS7) Peptide
category
Proteins and Peptides
provider
Abbexa
reference
abx616312
tested applications
P-ELISA
Description
NADH:Ubiquinone Oxidoreductase Core Subunit S7 (NDUFS7) Peptide is a synthetic peptide. This peptide is used in the production of
Documents del producto
Instrucciones
Data sheet
Product specifications
| Category | Proteins and Peptides |
| Immunogen Target | NADH:Ubiquinone Oxidoreductase Core Subunit S7 (NDUFS7) |
| Host | Synthetic |
| Assay Type | Activity: Not tested. Sequence Fragment: Internal region: C-SRGEYVVAKLD |
| Recommended Dilution | BL (predicted): 0.5 mg/ml. Optimal dilutions/concentrations should be determined by the end user. |
| Conjugation | Unconjugated |
| Size 1 | 100 µg |
| Form | Lyophilized |
| Tested Applications | P-ELISA |
| Buffer | Prior to lyophilization: Deionized water. |
| Availability | Shipped within 5-10 working days. |
| Storage | Aliquot and store at -20 °C. Avoid repeated freeze/thaw cycles. |
| Dry Ice | No |
| Gene ID | 374291, 75406, 362837 |
| NCBI Accession | NP_077718.3 |
| Background | Protein NDUFS7 |
| Status | RUO |
| Note | THIS PRODUCT IS FOR RESEARCH USE ONLY. NOT FOR USE IN DIAGNOSTIC, THERAPEUTIC OR COSMETIC PROCEDURES. NOT FOR HUMAN OR ANIMAL CONSUMPTION. Reconstitute in deionized water. |
Descripción
Related Products
Human NDUFS7 (NADH dehydrogenase [ubiquinone] iron-sulfur protein 7, mitochondrial) ELISA Kit
Ver Producto
NDUFS7 antibody
This gene encodes a protein that is a subunit of one of the complexes that forms the mitochondrial respiratory chain. This protein is one of over 40 subunits found in complex I, the nicotinamide adenine dinucleotide (NADH):ubiquinone oxidoreductase. This complex functions in the transfer of electrons from NADH to the respiratory chain, and ubiquinone is believed to be the immediate electron acceptor for the enzyme. Mutations in this gene cause Leigh syndrome due to mitochondrial complex I deficiency, a severe neurological disorder that results in bilaterally symmetrical necrotic lesions in subcortical brain regions.
Ver Producto