Frizzled Class Receptor 4 (FZD4) Antibody

Este producto es parte de FZD - Frizzled Homolog
Frizzled Class Receptor 4 (FZD4) Antibody
312€ (0.1 mg)

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Name
Frizzled Class Receptor 4 (FZD4) Antibody
Category
Primary Antibodies
Provider
Abbexa
Reference
abx140624
Tested Applications
FCM

Description

Frizzled Class Receptor 4 (FZD4) Antibody is a mouse monoclonal antibody against Frizzled Class Receptor 4 (FZD4).

Documentos del producto

Instrucciones
Data sheet
Descargar

Especificaciones del producto

Category
Primary Antibodies
Immunogen Target
Target: Frizzled Class Receptor 4 (FZD4)
Host
Mouse
Reactivity
Human, Monkey
Assay Type
Concentration: 1 mg/ml
Recommended Dilution
Optimal dilutions/concentrations should be determined by the end user.
Clonality
Monoclonal
Conjugation
Unconjugated
Isotype
IgG1 Kappa
Clone ID
F950
Purification
Purified by Protein A affinity chromatography.
Size 1
0.1 mg
Tested Applications
FCM
Buffer
PBS solution with 15 mM sodium azide.
Availability
Shipped within 5-12 working days.
Storage
Store at 2-8°C. Do not freeze.
Dry Ice
No
UniProt ID
Q9ULV1
Gene ID
8322
Alias
FZD4,Fz4,EVR1,FEVR,Fz-4,FzE4,GPC, hFz4,CD344,FZD4S,frizzled receptor 4
Background
Antibody anti-FZD4
Status
RUO
Note
THIS PRODUCT IS FOR RESEARCH USE ONLY. NOT FOR USE IN DIAGNOSTIC, THERAPEUTIC OR COSMETIC PROCEDURES. NOT FOR HUMAN OR ANIMAL CONSUMPTION.

Background

FZD4 is a Wnt receptor that mediates canonical β-catenin-dependent signaling and non-canonical pathways. It is expressed in vascular endothelial cells, the retina, and skeletal tissue, playing a key role in angiogenesis, vascular integrity, and retinal development. FZD4 interacts with Wnt ligands and coreceptors like LRP5/6 to activate β-catenin signaling, which is essential for retinal vascularization and blood-retina barrier maintenance. Mutations in FZD4 are associated with familial exudative vitreoretinopathy (FEVR), a genetic disorder characterized by incomplete retinal vascular development, leading to vision loss. In cancer, FZD4 promotes tumor angiogenesis and metastasis by activating Wnt signaling pathways that support endothelial cell proliferation and survival. Its overexpression has been observed in breast and colorectal cancers, where it enhances angiogenesis and therapy resistance. FZD4 also regulates bone formation and osteoblast differentiation, contributing to skeletal development. Targeting FZD4 and its signaling pathways has therapeutic potential in vascular disorders, cancer, and ocular diseases. Overall, FZD4 is essential for vascular development and maintenance, with dysregulation contributing to angiogenic disorders and tumor progression.

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