PLSCR4 antibody
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Description
May mediate accelerated ATP-independent bidirectional transbilayer migration of phospholipids upon binding calcium ions that results in a loss of phospholipid asymmetry in the plasma membrane. May play a central role in the initiation of fibrin clot formation, in the activation of mast cells and in the recognition of apoptotic and injured cells by the reticuloendothelial system.
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Product specifications
| Category | Primary Antibodies |
| Immunogen Target | phospholipid scramblase 4 (PLSCR4) |
| Host | Rabbit |
| Reactivity | Human, Mouse, Rat |
| Recommended Dilution | WB: 1:500-1:2000 |
| Clonality | polyclonal |
| Conjugation | Unconjugated |
| Isotype | IgG |
| Observed MW | 37 kDa |
| Purity | ≥95% as determined by SDS-PAGE |
| Purification | Immunogen affinity purified |
| Size 1 | 100µg |
| Form | liquid |
| Tested Applications | ELISA, WB |
| Storage | PBS with 0.02% sodium azide and 50% glycerol pH 7.3, -20℃ for 12 months(Avoid repeated freeze / thaw cycles.) |
| UniProt ID | Q9NRQ2 |
| Gene ID | 57088 |
| Alias | Phospholipid scramblase 4 (PL scramblase 4),Ca(2+)-dependent phospholipid scramblase 4,Cell growth-inhibiting gene 43 protein,TRA1,PLSCR4 |
| Background | Antibody anti-PLSCR4 |
| Status | RUO |
| Note | Mol. Weight 37 kDa |
Descripción
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PLSCR4 antibody
May mediate accelerated ATP-independent bidirectional transbilayer migration of phospholipids upon binding calcium ions that results in a loss of phospholipid asymmetry in the plasma membrane. May play a central role in the initiation of fibrin clot formation, in the activation of mast cells and in the recognition of apoptotic and injured cells by the reticuloendothelial system.
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